Marie Curie
This project is funded
by the European Union

Marta Artal- Sanz


Research positions

From March 2012) Ramón y Cajal Researcher. University Pablo de Olavide, Seville, Spain.
Andalusian Center for Developmental Biology

(2011-2012) Group leader. University of Freiburg, Germany.
Institute for Biology (Prof. Dr. Ralf Baumeister)

(2008-2011) Juan de la Cierva Post-doctoral Fellow.
IBV, CSIC, Valencia, Spain (Prof. Dr Francesc Palau)
“Establishing Caenorhabditis elegans as a model system to study mitochondrial neurodegenerative disorders”

(2007-2008) Postdoctoral Fellow
European Programme LSHG-CT-2004-511983, TRANSDEATH. Institute of Molecular Biology and Biotechnology, Crete, Greece (Prof. Dr. N. Tarvernarakis)

(2005-2007) Marie Curie Postdoctoral Fellowship
Institute of Molecular Biology and Biotechnology, Crete, Greece (Prof. Dr. N. Tarvernarakis)
“Mitochondrial pathways in neurodegeneration_DeMiSe”

(1999-2003) PhD student. University of Amsterdam, The Netherlands
Swammerdam Institute of Life Sciences (Prof. Dr. Les Grivell)
“The role of the PHB complex in mitochondrial biogenesis. Structural and functional studies in Saccharomyces cerevisiae and Caenorhabditis elegans


  • A high-throughput method for the analysis of larval developmental phenotypes in Caenorhabditis elegans.
  • Olmedo M§, M Geibel, M Artal-Sanz and M Merrow§ (§ Corresponding authors).
  • Genetics, 2015 201: 443-448 
  • Analysis of the effect of the mitochondrial prohibitin complex, a context-dependent modulator of longevity, on the C. elegans metabolome.
  • Lourenço AB, Muñoz-Jiménez C, Venegas-Calerón M, Artal-Sanz M.
  • Biochim Biophys Acta, 2015, 1847(11):1457-68.
  • Prohibitin-Mediated Lifespan and Mitochondrial Stress Implicate SGK-1, Insulin/IGF and mTORC2 in C. elegans.
  • Gatsi, R., B. Schulze, M.J. Rodríguez-Palero, B. Hernando-Rodríguez, R. Baumeister and M. Artal-Sanz..
  • PLoS One. 2014, 9 - 9, pp. e107671.
  • Opposing function of mitochondrial prohibitin in aging.
  • Artal-Sanz M*, and N. Tavernarakis.
  • Aging (Albany NY) 2010, 2: 1004-11 (*Corresponding author). 
  • Prohibitin couples diapause signalling to mitochondrial metabolism during ageing in C. elegans.
  • Artal-Sanz M*, and N. Tavernarakis.
  • Nature, 2009, 461: 793-7 (*Corresponding author)
  • Prohibitin and mitochondrial biology.
  • Artal-Sanz M, and N. Tavernarakis.
  • TRENDS in Endocrinology and Metabolism, 2009, 20: 394-401.
  • Lysosomal biogenesis and function is critical for necrotic cell death in Caenorhabditis elegans.
  • Artal-Sanz M, et al.
  • J Cell Biol, 2006, 173: 231-9.
  • Caenorhabditis elegans: a versatile platform for drug discovery.
  • Artal-Sanz M, et al. 2006.
  • Biotechnol J, 2006, 1: 1405-18.
  • The mitochondrial prohibitin complex is essential for embryonic viability and germline function in Caenorhabditis elegans.
  • Artal-Sanz M*, et al.
  • J Biol Chem, 22003, 278: 32091-9. (*Corresponding author).
  • Krijgsveld J, R.F. Ketting, T. Mahmoudi, J. Johansen, M. Artal-Sanz et al.
  • Metabolic labeling of C. elegans and D. melanogaster for quantitative proteomics.
  • Nat Biotechnol, 2003, 21: 927-31.
  • Prohibitins act as a membrane-bound chaperone for the stabilization of mitochondrial proteins.
  • Nijtmans LG*, L. de Jong*, M. Artal-Sanz*, et al.
  • EMBO J, 200, 19: 2444-51 (*Equal contribution)